Avian sarcoma and leukosis virus gag gene - Genet. Mol. Res. 14 (4): 14379-14386 “Avian sarcoma and leukosis virus gag gene in the Anser anser domesticus genome”

D. Elleder, Hejnar, J., Elleder, D., Hejnar, J., Elleder, D., and Hejnar, J., Avian sarcoma and leukosis virus gag gene - Genet. Mol. Res. 14 (4): 14379-14386 “Avian sarcoma and leukosis virus gag gene in the Anser anser domesticus genome”, vol. 15. p. -, 2016.

Dear Editor, A recent paper in the GMR Journal (Zhu et al., 2015) reported the discovery of endogenous avian sarcoma and leukosis virus (enASLV) in the domestic goose (Anser anser domesticus) genome. The discovery was based on PCR detection of a single viral gene (gag). This would be a very surprising finding, as ASLV-related endogenous retroviruses have only been detected in galliform birds (Dimcheff et al., 2000). Geese, which belong to the order Anseriformes, split from the Galliformes more than 80 million years ago.

Accurate description of phages and their genomes - Genet. Mol. Res. 14 (1): 190-198 “Bioinformatic analysis of phage AB3, a phiKMV-like virus infecting Acinetobacter baumannii”

A. M. Kropinski, Accurate description of phages and their genomes - Genet. Mol. Res. 14 (1): 190-198 “Bioinformatic analysis of phage AB3, a phiKMV-like virus infecting Acinetobacter baumannii”, vol. 14. pp. 15902-15903, 2015.

Dear Editor, By way of introduction, I have been reviewing bacteriophage manuscripts for 40 years, I am a RefSeq Genome Advisor (phages) to NCBI, and the current chair of the Bacterial and Archaeal Viruses Subcommittee of the International Committee on Viruses Taxonomy. Therefore, I am very interested in accurate published descriptions of phages and their genomes.

Atorvastatin in combination with ezetimibe and carotid atherosclerosis

K. I. Paraskevas, Mikhailidis, D. P., and Giannoukas, A. D., Atorvastatin in combination with ezetimibe and carotid atherosclerosis, vol. 13. pp. 4805-4807, 2014.

Dear Editor, Luo et al. (2014) reported that the combination of atorvastatin plus ezetimibe de­creased carotid intima media thickness (cIMT) significantly more than atorvastatin mono­therapy. We would like to add a few comments. The ENHANCE trial (Kastelein et al., 2008) did not compare the effect of atorvastatin plus ezetimibe versus atorvastatin monotherapy on cIMT, as mentioned by Luo et al. (2014). The statin used was simvastatin. As Luo et al. (2014) state, the cIMT in ENHANCE was set too low and could not be reduced further.

Reply to commentary by A. Fisette on the article “Relationship between the acylation-stimulating protein gene and coronary heart disease in the Xinjiang Uygur and Han populations of China” published in Genetics and Molecular Research 13 (2): 2638-2644 (2014) to the letter published in Genet. Mol. Res. 13 (4): 9134-9135

Y. Chen and Ma, Y. T., Reply to commentary by A. Fisette on the article “Relationship between the acylation-stimulating protein gene and coronary heart disease in the Xinjiang Uygur and Han populations of China” published in Genetics and Molecular Research 13 (2): 2638-2644 (2014) to the letter published in Genet. Mol. Res. 13 (4): 9134-9135, vol. 13. pp. 9136-9137, 2014.

Dear Editor, In answer to the questioning of the terminology we used in our manuscript (Chen et al., 2014), ASP is a 76-amino acid (8932 Da) fragment, which is identical to C3adesArg, a cleavage product of complement C3. Cleavage of complement C3 is mediated via the alter­nate complement pathway by the interaction of C3, factor B and adipsin (Factor D, a serine protease enzyme), which generates C3a. Rapid cleavage of the C terminal arginine of C3a by carboxypeptidase N generates ASP (Hugli, 1990).

"Relationship between the acylation-stimulating protein gene and coronary heart disease in the Xinjiang Uygur and Han populations of China", published in Genetics and Molecular Research 13 (2): 2638-2644 (2014)

A. Fisette, "Relationship between the acylation-stimulating protein gene and coronary heart disease in the Xinjiang Uygur and Han populations of China", published in Genetics and Molecular Research 13 (2): 2638-2644 (2014), vol. 13. pp. 9134-9135, 2014.

Dear Editor, As a scientist who works on obesity and immunity, especially the complement system, I read the paper titled "Relationship between the acylation-stimulating protein gene and coro­nary heart disease in the Xinjiang Uygur and Han populations of China" published in Genetics and Molecular Research 13 (2): 2638-2644 (2014). I was surprised when I read about the "acylation-stimulating protein (ASP) gene" and its correlation with heart disease.

RETRACTION of "Effects of destrin pathway mutations on the gene expression profile" by J.N. Xiu, X. Liu, H. Wang, C.M. Hu, Q.H. Luo and Q.Q. Zhou. Genet. Mol. Res. 13 (2): 2628-2637 (2014)

J. N. Xu, Liu, X., Wang, H., Hu, C. M., Luo, Q. H., and Zhou, Q. Q., RETRACTION of "Effects of destrin pathway mutations on the gene expression profile" by J.N. Xiu, X. Liu, H. Wang, C.M. Hu, Q.H. Luo and Q.Q. Zhou. Genet. Mol. Res. 13 (2): 2628-2637 (2014), vol. 13. pp. 10599-10599, 2014.

This paper has been retracted with the consent of the submitting author, Hui Wang because of serious plagiary issues. The same article was previously published in the African Journal of Pharmacy and Pharmacology vol. 6 (24), pp. 1746-1752 in 2012, DOI: 10.5897/AJPP12.067; the authors were Qinghua Zhou, Yun Li and Baihua Chen. The republication of this article is a serious breach of scientific ethics. After Dr. Wang was informed of this situation; he agreed that the paper should be retracted. We also contacted Dr. Wang’s institution, a University Hospital in China.

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