Publications
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“Meta-analysis demonstrates association between Arg72Pro polymorphism in the P53 gene and susceptibility to keloids in the Chinese population”, vol. 11, pp. 1701-1711, 2012.
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Al-Attar A, Mess S, Thomassen JM, Kauffman CL, et al. (2006). Keloid pathogenesis and treatment. Plast. Reconstr. Surg. 117: 286-300.
http://dx.doi.org/10.1097/01.prs.0000195073.73580.46
PMid:16404281
Atiyeh BS, Costagliola M and Hayek SN (2005). Keloid or hypertrophic scar: the controversy: review of the literature. Ann. Plast. Surg. 54: 676-680.
http://dx.doi.org/10.1097/01.sap.0000164538.72375.93
PMid:15900161
Bayat A, McGrouther DA and Ferguson MW (2003). Skin scarring. BMJ 326: 88-92.
http://dx.doi.org/10.1136/bmj.326.7380.88
PMid:12521975 PMCid:1125033
Butler PD, Longaker MT and Yang GP (2008). Current progress in keloid research and treatment. J. Am. Coll. Surg. 206: 731-741.
http://dx.doi.org/10.1016/j.jamcollsurg.2007.12.001
PMid:18387480
De Felice B, Ciarmiello LF, Mondola P, Damiano S, et al. (2007). Differential p63 and p53 expression in human keloid fibroblasts and hypertrophic scar fibroblasts. DNA Cell Biol. 26: 541-547.
http://dx.doi.org/10.1089/dna.2007.0591
PMid:17688405
Higgins JP and Thompson SG (2002). Quantifying heterogeneity in a meta-analysis. Stat. Med. 21: 1539-1558.
http://dx.doi.org/10.1002/sim.1186
PMid:12111919
Jin J, Gao JH and Lu F (2007). Clinical experiment of susceptible people to keloid. Zhongguo Lin Chuang Jie Pao Xue Za Zhi 25: 320-322.
Ladin DA, Hou Z, Patel D, McPhail M, et al. (1998). p53 and apoptosis alterations in keloids and keloid fibroblasts. Wound Repair Regen. 6: 28-37.
http://dx.doi.org/10.1046/j.1524-475X.1998.60106.x
PMid:9776848
Liu Y (2007). Preliminary Linkage Analysis of Keloid Susceptibility Loci and Polymorphisms of Correlation Genes in Chinese Han Population. Master's thesis, China Medical University, Shenyang.
Liu YB (2008). The Study of Impaired Apoptosis Function of Fas and P53 Protein in the Fibroblasts Derived from Keloid. PhD thesis, Southern Medical University, Guangzhou.
Liu YB, Gao JH, Duan HJ and Liu XJ (2003). Investigation of p53 gene mutations in keloids using PCR-SSCP. Zhonghua Zheng Xing Wai Ke Za Zhi 19: 258-260.
PMid:14628411
Liu W, Jiang YH, Li YL, Lin ZH, et al. (2004). Experimental study on p53 gene mutation in keloid fibroblasts. Zhonghua Shao Shang Za Zhi 20: 85-87.
PMid:15312469
Marneros AG and Krieg T (2004). Keloids-clinical diagnosis, pathogenesis, and treatment options. J. Dtsch. Dermatol. Ges. 2: 905-913.
http://dx.doi.org/10.1046/j.1439-0353.2004.04077.x
PMid:16281608
Matlashewski GJ, Tuck S, Pim D, Lamb P, et al. (1987). Primary structure polymorphism at amino acid residue 72 of human p53. Mol. Cell Biol. 7: 961-963.
PMid:3547088 PMCid:365159
McGregor JM, Harwood CA, Brooks L, Fisher SA, et al. (2002). Relationship between p53 codon 72 polymorphism and susceptibility to sunburn and skin cancer. J. Invest. Dermatol. 119: 84-90.
http://dx.doi.org/10.1046/j.1523-1747.2002.01655.x
PMid:12164929
Menezes HL, Juca MJ, Gomes EG, Nunes BL, et al. (2010). Analysis of the immunohistochemical expressions of p53, bcl-2 and Ki-67 in colorectal adenocarcinoma and their correlations with the prognostic factors. Arq. Gastroenterol. 47: 141-147.
http://dx.doi.org/10.1590/S0004-28032010000200005
PMid:20721457
Peters JL, Sutton AJ, Jones DR, Abrams KR, et al. (2006). Comparison of two methods to detect publication bias in meta-analysis. JAMA 295: 676-680.
http://dx.doi.org/10.1001/jama.295.6.676
PMid:16467236
Pezeshki A, Sari-Aslani F, Ghaderi A and Doroudchi M (2006). p53 codon 72 polymorphism in basal cell carcinoma of the skin. Pathol. Oncol. Res. 12: 29-33.
http://dx.doi.org/10.1007/BF02893428
PMid:16554913
Saed GM, Ladin D, Olson J, Han X, et al. (1998). Analysis of p53 gene mutations in keloids using polymerase chain reaction-based single-strand conformational polymorphism and DNA sequencing. Arch. Dermatol. 134: 963-967.
http://dx.doi.org/10.1001/archderm.134.8.963
PMid:9722726
Sakamuro D, Sabbatini P, White E and Prendergast GC (1997). The polyproline region of p53 is required to activate apoptosis but not growth arrest. Oncogene 15: 887-898.
http://dx.doi.org/10.1038/sj.onc.1201263
PMid:9285684
Sayah DN, Soo C, Shaw WW, Watson J, et al. (1999). Downregulation of apoptosis-related genes in keloid tissues. J. Surg. Res. 87: 209-216.
http://dx.doi.org/10.1006/jsre.1999.5761
PMid:10600351
Sjalander A, Birgander R, Kivela A and Beckman G (1995). p53 polymorphisms and haplotypes in different ethnic groups. Hum. Hered. 45: 144-149.
http://dx.doi.org/10.1159/000154275
PMid:7615299
Tanaka A, Hatoko M, Tada H, Iioka H, et al. (2004). Expression of p53 family in scars. J. Dermatol. Sci. 34: 17-24.
http://dx.doi.org/10.1016/j.jdermsci.2003.09.005
PMid:14757278
Teofoli P, Barduagni S, Ribuffo M, Campanella A, et al. (1999). Expression of Bcl-2, p53, c-jun and c-fos protooncogenes in keloids and hypertrophic scars. J. Dermatol. Sci. 22: 31-37.
http://dx.doi.org/10.1016/S0923-1811(99)00040-7
Thomas M, Kalita A, Labrecque S, Pim D, et al. (1999). Two polymorphic variants of wild-type p53 differ biochemically and biologically. Mol. Cell Biol. 19: 1092-1100.
PMid:9891044 PMCid:116039
Vandenbroucke JP, von Elm E, Altman DG, Gotzsche PC, et al. (2007). Strengthening the Reporting of Observational Studies in Epidemiology (STROBE): explanation and elaboration. Epidemiology 18: 805-835.
http://dx.doi.org/10.1097/EDE.0b013e3181577511
PMid:18049195
Walker KK and Levine AJ (1996). Identification of a novel p53 functional domain that is necessary for efficient growth suppression. Proc. Natl. Acad. Sci. U. S. A. 93: 15335-15340.
http://dx.doi.org/10.1073/pnas.93.26.15335
PMid:8986812 PMCid:26405
Wang CM, Hiko H and Nakazawa N (2005). Investigation of p53 polymorphism for genetic predisposition of keloid and hypertrophic scar. Zhonghua Zheng Xing Wai Ke Za Zhi 21: 32-35.
PMid:15844595
Yan L, Lu XY, Wang CM, Cao R, et al. (2007). Association between p53 gene codon 72 polymorphism and keloid in Chinese population. Zhonghua Zheng Xing Wai Ke Za Zhi 23: 428-430.
PMid:18161363
Zhuo Y, Gao JH, Luo SQ, Zeng WS, et al. (2005). p53 gene codon 72 polymorphism and susceptibility to keloid. Zhonghua Zheng Xing Wai Ke Za Zhi 21: 201-203.
PMid:16128105
Zhuo Y, Gao JH and Zeng XY (2008). The application of P53 gene detection kit for susceptibility of keloid. Zhongguo Mei Rong Yi Xue 5: 694-696.
Zintzaras E and Ioannidis JP (2005). Heterogeneity testing in meta-analysis of genome searches. Genet. Epidemiol. 28: 123-137.
http://dx.doi.org/10.1002/gepi.20048
PMid:15593093
“Association of -619C/T polymorphism in CDSN gene and psoriasis risk: a meta-analysis”, vol. 10, pp. 3632-3640, 2011.
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Ahnini RT, Camp NJ, Cork MJ, Mee JB, et al. (1999). Novel genetic association between the corneodesmosin (MHC S) gene and susceptibility to psoriasis. Hum. Mol. Genet. 8: 1135-1140.
http://dx.doi.org/10.1093/hmg/8.6.1135
Ameen M, Allen MH, Fisher SA, Lewis CM, et al. (2005). Corneodesmosin (CDSN) gene association with psoriasis vulgaris in Caucasian but not in Japanese populations. Clin. Exp. Dermatol. 30: 414-418.
http://dx.doi.org/10.1111/j.1365-2230.2005.01789.x
PMid:15953084
Asumalahti K, Laitinen T, Itkonen-Vatjus R, Lokki ML, et al. (2000). A candidate gene for psoriasis near HLA-C, HCR (Pg8), is highly polymorphic with a disease-associated susceptibility allele. Hum. Mol. Genet. 9: 1533-1542.
http://dx.doi.org/10.1093/hmg/9.10.1533
PMid:10888604
Attia J, Thakkinstian A and D'Este C (2003). Meta-analyses of molecular association studies: methodologic lessons for genetic epidemiology. J. Clin. Epidemiol. 56: 297-303.
http://dx.doi.org/10.1016/S0895-4356(03)00011-8
Genetics and Molecular Research 10 (4): 3632-3640 (2011) Butt C, Rahman P, Siannis F, Farewell VT, et al. (2005). Corneodesmosin polymorphisms in psoriatic arthritis. Rheumatology 44: 684-685.
Capon F, Munro M, Barker J and Trembath R (2002). Searching for the major histocompatibility complex psoriasis susceptibility gene. J. Invest. Dermatol. 118: 745-751.
http://dx.doi.org/10.1046/j.1523-1747.2002.01749.x
PMid:11982750
Capon F, Toal IK, Evans JC, Allen MH, et al. (2003). Haplotype analysis of distantly related populations implicates corneodesmosin in psoriasis susceptibility. J. Med. Genet. 40: 447-452.
http://dx.doi.org/10.1136/jmg.40.6.447
PMid:12807967 PMCid:1735499
Caubet C, Jonca N, Brattsand M, Guerrin M, et al. (2004). Degradation of corneodesmosome proteins by two serine proteases of the kallikrein family, SCTE/KLK5/hK5 and SCCE/KLK7/hK7. J. Invest. Dermatol. 122: 1235-1244.
http://dx.doi.org/10.1111/j.0022-202X.2004.22512.x
PMid:15140227
Chang YT, Tsai SF, Lin MW, Liu HN, et al. (2003). SPR1 gene near HLA-C is unlikely to be a psoriasis susceptibility gene. Exp. Dermatol. 12: 307-314.
http://dx.doi.org/10.1034/j.1600-0625.2003.00039.x
PMid:12823445
Chang YT, Chou CT, Shiao YM, Lin MW, et al. (2006). Psoriasis vulgaris in Chinese individuals is associated with PSORS1C3 and CDSN genes. Br. J. Dermatol. 155: 663-669.
http://dx.doi.org/10.1111/j.1365-2133.2006.07420.x
PMid:16965413
Griffiths CE and Barker JN (2007). Pathogenesis and clinical features of psoriasis. Lancet 370: 263-271.
http://dx.doi.org/10.1016/S0140-6736(07)61128-3
Guerrin M, Simon M, Montezin M, Haftek M, et al. (1998). Expression cloning of human corneodesmosin proves its identity with the product of the S gene and allows improved characterization of its processing during keratinocyte differentiation. J. Biol. Chem. 273: 22640-22647.
http://dx.doi.org/10.1074/jbc.273.35.22640
PMid:9712893
Higgins JP and Thompson SG (2002). Quantifying heterogeneity in a meta-analysis. Stat. Med. 21: 1539-1558.
http://dx.doi.org/10.1002/sim.1186
PMid:12111919
Hui J, Oka A, Tamiya G, Tomizawa M, et al. (2002). Corneodesmosin DNA polymorphisms in MHC haplotypes and Japanese patients with psoriasis. Tissue Antigens 60: 77-83.
http://dx.doi.org/10.1034/j.1399-0039.2002.600110.x
PMid:12366786
Krueger JG, Krane JF, Carter DM and Gottlieb AB (1990). Role of growth factors, cytokines, and their receptors in the pathogenesis of psoriasis. J. Invest. Dermatol. 94: 135S-140S.
http://dx.doi.org/10.1111/1523-1747.ep12876121
PMid:2161887
Liu Y, Krueger JG and Bowcock AM (2007). Psoriasis: genetic associations and immune system changes. Genes Immun. 8: 1-12.
http://dx.doi.org/10.1038/sj.gene.6364351
PMid:17093502
O'Brien KP, Holm SJ, Nilsson S, Carlen L, et al. (2001). The HCR gene on 6p21 is unlikely to be a psoriasis susceptibility gene. J. Invest. Dermatol. 116: 750-754.
http://dx.doi.org/10.1046/j.0022-202x.2001.01323.x
PMid:11348465
Orrù S, Giuressi E, Casula M, Loizedda A, et al. (2002). Psoriasis is associated with a SNP haplotype of the corneodesmosin gene (CDSN). Tissue Antigens 60: 292-298.
http://dx.doi.org/10.1034/j.1399-0039.2002.600403.x
PMid:12472658
Peters JL, Sutton AJ, Jones DR, Abrams KR, et al. (2006). Comparison of two methods to detect publication bias in metaanalysis. JAMA 295: 676-680.
http://dx.doi.org/10.1001/jama.295.6.676
PMid:16467236
Romphruk AV, Oka A, Romphruk A, Tomizawa M, et al. (2003). Corneodesmosin gene: no evidence for PSORS 1 gene in North-eastern Thai psoriasis patients. Tissue Antigens 62: 217-224.
http://dx.doi.org/10.1034/j.1399-0039.2003.00056.x
PMid:12956875
Schmitt-Egenolf M, Windemuth C, Hennies HC, Albis-Camps M, et al. (2001). Comparative association analysis reveals that corneodesmosin is more closely associated with psoriasis than HLA-Cw*0602-B*5701 in German families. Tissue Antigens 57: 440-446.
http://dx.doi.org/10.1034/j.1399-0039.2001.057005440.x
PMid:11556968
Terui T, Ozawa M and Tagami H (2000). Role of neutrophils in induction of acute inflammation in T-cell-mediated immune dermatosis, psoriasis: a neutrophil-associated inflammation-boosting loop. Exp. Dermatol. 9: 1-10.
http://dx.doi.org/10.1034/j.1600-0625.2000.009001001.x
PMid:10688368
Vandenbroucke JP, von EE, Altman DG, Gotzsche PC, et al. (2007). Strengthening the reporting of observational studies in epidemiology (STROBE): explanation and elaboration. Epidemiology 18: 805-835.
http://dx.doi.org/10.1097/EDE.0b013e3181577511
PMid:18049195
Zenz R, Eferl R, Kenner L, Florin L, et al. (2005). Psoriasis-like skin disease and arthritis caused by inducible epidermal deletion of Jun proteins. Nature 437: 369-375.
http://dx.doi.org/10.1038/nature03963
PMid:16163348
Zintzaras E and Loannidis JP (2005). Heterogeneity testing in meta-analysis of genome searches. Genet. Epidemiol. 28: 123-137.
http://dx.doi.org/10.1002/gepi.20048
PMid:15593093