Publications
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“APOA5 polymorphisms associated with lipid metabolism in Brazilian children and adolescents”, vol. 15, p. -, 2016.
, , , “Influence of the 48867A>C (Asp358Ala) IL6R polymorphism on response to a lifestyle modification intervention in individuals with metabolic syndrome”, vol. 12, pp. 3983-3991, 2013.
, “More severe clinical course of cardiovascular dysfunction in intensive care unit patients with the 894TT eNOS genotype”, vol. 12, pp. 562-568, 2013.
, Antoniades C, Tousoulis D, Vasiliadou C, Pitsavos C, et al. (2005). Genetic polymorphism on endothelial nitric oxide synthase affects endothelial activation and inflammatory response during the acute phase of myocardial infarction. J. Am. Coll. Cardiol. 46: 1101-1109.
http://dx.doi.org/10.1016/j.jacc.2005.05.072
PMid:16168297
Dafni C, Drakoulis N, Landt O, Panidis D, et al. (2010). Association of the eNOS E298D polymorphism and the risk of myocardial infarction in the Greek population. BMC Med. Genet. 11: 133.
http://dx.doi.org/10.1186/1471-2350-11-133
PMid:20854685 PMCid:2954842
Fallavena PRV, Borges TJ, Paskulin DD, Thurow HS, et al. (2013). The synergy of -260T T CD14 and -308GG TNF-α genotypes in survival of critically ill patients. Scandinav. J. Immunol. 77: 62-68.
http://dx.doi.org/10.1111/sji.12002
PMid:23036097
Ferreira FL, Bota DP, Bross A, Melot C, et al. (2001). Serial evaluation of the SOFA score to predict outcome in critically ill patients. JAMA 286: 1754-1758.
http://dx.doi.org/10.1001/jama.286.14.1754
PMid:11594901
Hibi K, Ishigami T, Tamura K, Mizushima S, et al. (1998). Endothelial nitric oxide synthase gene polymorphism and acute myocardial infarction. Hypertension 32: 521-526.
http://dx.doi.org/10.1161/01.HYP.32.3.521
PMid:9740620
Hingorani AD, Liang CF, Fatibene J, Lyon A, et al. (1999). A common variant of the endothelial nitric oxide synthase (Glu298→Asp) is a major risk factor for coronary artery disease in the UK. Circulation 100: 1515-1520.
http://dx.doi.org/10.1161/01.CIR.100.14.1515
PMid:10510054
Huttunen R, Hurme M, Laine J, Eklund C, et al. (2009). Endothelial nitric oxide synthase G894T (GLU298ASP) polymorphism is associated with hypotension in patients with E. coli bacteremia but not in bacteremia caused by a gram-positive organism. Shock 31: 448-453.
http://dx.doi.org/10.1097/SHK.0b013e318188e58e
PMid:18827745
Iturry-Yamamoto GR, Moriguchi EH, Zago AC, Alho C, et al. (2007). Association of the 894G>T polymorphism of the endothelial constitutive nitric oxide synthase gene with unstable angina. Braz. J. Med. Biol. Res. 40: 475-483.
http://dx.doi.org/10.1590/S0100-879X2006005000072
PMid:17401490
Kerkeni M, Addad F, Chauffert M, Myara A, et al. (2006). Hyperhomocysteinemia, endothelial nitric oxide synthase polymorphism, and risk of coronary artery disease. Clin. Chem. 52: 53-58.
http://dx.doi.org/10.1373/clinchem.2005.057950
PMid:16284093
Knaus WA, Draper EA, Wagner DP and Zimmerman JE (1985). APACHE II: a severity of disease classification system. Crit. Care Med. 13: 818-829.
http://dx.doi.org/10.1097/00003246-198510000-00009
PMid:3928249
Krishnagopalan S, Kumar A, Parrillo JE and Kumar A (2002). Myocardial dysfunction in the patient with sepsis. Curr. Opin. Crit. Care 8: 376-388.
http://dx.doi.org/10.1097/00075198-200210000-00003
PMid:12357104
Lahiri DK and Nurnberger JI Jr (1991). A rapid non-enzymatic method for the preparation of HMW DNA from blood for RFLP studies. Nucleic Acids Res. 19: 5444.
http://dx.doi.org/10.1093/nar/19.19.5444
PMid:1681511 PMCid:328920
Lembo G, De Luca N, Battagli C, Iovino G, et al. (2001). A common variant of endothelial nitric oxide synthase (Glu298Asp) is an independent risk factor for carotid atherosclerosis. Stroke 32: 735-740.
http://dx.doi.org/10.1161/01.STR.32.3.735
PMid:11239195
Levy MM, Fink MP, Marshall JC, Abraham E, et al. (2003). 2001 SCCM/ESICM/ACCP/ATS/SIS International sepsis definitions conference. Intensive Care Med. 29: 530-538.
PMid:12664219
Miyamoto Y, Saito Y, Kajiyama N, Yoshimura M, et al. (1998). Endothelial nitric oxide synthase gene is positively associated with essential hypertension. Hypertension 32: 3-8.
http://dx.doi.org/10.1161/01.HYP.32.1.3
PMid:9674630
Moncada S and Higgs A (1993). The L-arginine-nitric oxide pathway. N. Engl. J. Med. 329: 2002-2012.
http://dx.doi.org/10.1056/NEJM199312303292706
PMid:7504210
Nadaud S, Bonnardeaux A, Lathrop M and Soubrier F (1994). Gene structure, polymorphism and mapping of the human endothelial nitric oxide synthase gene. Biochem. Biophys. Res. Commun. 198: 1027-1033.
http://dx.doi.org/10.1006/bbrc.1994.1146
PMid:7509596
Paludo FJO, Picanço JB, Fallavena PRV, Fraga LR, et al. (2013). Higher frequency of septic shock in septic patients with the 47C allele (rs4880) of the SOD2 gene. Gene 517: 106-111.
http://dx.doi.org/10.1016/j.gene.2012.10.078
PMid:23142385
Parra FC, Amado RC, Lambertucci JR, Rocha J, et al. (2003). Color and genomic ancestry in Brazilians. Proc. Natl. Acad. Sci. U. S. A. 100: 177-182.
http://dx.doi.org/10.1073/pnas.0126614100
PMid:12509516 PMCid:140919
Paskulin DD, Fallavena PRV, Paludo FJO, Borges TJ, et al. (2011). TNF -308G > a promoter polymorphism (rs1800629) and outcome from critical illness. Braz. J. Infect. Dis. 15: 231-238.
http://dx.doi.org/10.1016/S1413-8670(11)70181-7
Tesauro M, Thompson WC, Rogliani P, Qi L, et al. (2000). Intracellular processing of endothelial nitric oxide synthase isoforms associated with differences in severity of cardiopulmonary diseases: cleavage of proteins with aspartate vs. glutamate at position 298. Proc. Natl. Acad. Sci. U. S. A. 97: 2832-2835.
http://dx.doi.org/10.1073/pnas.97.6.2832
PMid:10717002 PMCid:16015
Umans JG and Levi R (1995). Nitric oxide in the regulation of blood flow and arterial pressure. Annu. Rev. Physiol. 57: 771-790.
http://dx.doi.org/10.1146/annurev.ph.57.030195.004011
PMid:7539996
Veldman BA, Spiering W, Doevendans PA, Vervoort G, et al. (2002). The Glu298Asp polymorphism of the NOS 3 gene as a determinant of the baseline production of nitric oxide. J. Hypertens. 20: 2023-2027.
http://dx.doi.org/10.1097/00004872-200210000-00022
PMid:12359981
Vincent JL, de Mendonça A, Cantraine F, Moreno R, et al. (1998). Use of the SOFA score to assess the incidence of organ dysfunction/failure in intensive care units: results of a multicenter, prospective study. Working group on "sepsis-related problems" of the European Society of Intensive Care Medicine. Crit. Care Med. 26: 1793-1800.
http://dx.doi.org/10.1097/00003246-199811000-00016
PMid:9824069