Publications

Found 2 results
Filters: Author is Y.J. Luo  [Clear All Filters]
2012
Y. Wu, Wang, B., Li, Y. H., Xu, X. G., Luo, Y. J., Chen, J. Z. S., Wei, H. C., Gao, X. H., and Chen, H. D., Meta-analysis demonstrates association between Arg72Pro polymorphism in the P53 gene and susceptibility to keloids in the Chinese population, vol. 11, pp. 1701-1711, 2012.
Al-Attar A, Mess S, Thomassen JM, Kauffman CL, et al. (2006). Keloid pathogenesis and treatment. Plast. Reconstr. Surg. 117: 286-300. http://dx.doi.org/10.1097/01.prs.0000195073.73580.46 PMid:16404281   Atiyeh BS, Costagliola M and Hayek SN (2005). Keloid or hypertrophic scar: the controversy: review of the literature. Ann. Plast. Surg. 54: 676-680. http://dx.doi.org/10.1097/01.sap.0000164538.72375.93 PMid:15900161   Bayat A, McGrouther DA and Ferguson MW (2003). Skin scarring. BMJ 326: 88-92. http://dx.doi.org/10.1136/bmj.326.7380.88 PMid:12521975 PMCid:1125033   Butler PD, Longaker MT and Yang GP (2008). Current progress in keloid research and treatment. J. Am. Coll. Surg. 206: 731-741. http://dx.doi.org/10.1016/j.jamcollsurg.2007.12.001 PMid:18387480   De Felice B, Ciarmiello LF, Mondola P, Damiano S, et al. (2007). Differential p63 and p53 expression in human keloid fibroblasts and hypertrophic scar fibroblasts. DNA Cell Biol. 26: 541-547. http://dx.doi.org/10.1089/dna.2007.0591 PMid:17688405   Higgins JP and Thompson SG (2002). Quantifying heterogeneity in a meta-analysis. Stat. Med. 21: 1539-1558. http://dx.doi.org/10.1002/sim.1186 PMid:12111919   Jin J, Gao JH and Lu F (2007). Clinical experiment of susceptible people to keloid. Zhongguo Lin Chuang Jie Pao Xue Za Zhi 25: 320-322.   Ladin DA, Hou Z, Patel D, McPhail M, et al. (1998). p53 and apoptosis alterations in keloids and keloid fibroblasts. Wound Repair Regen. 6: 28-37. http://dx.doi.org/10.1046/j.1524-475X.1998.60106.x PMid:9776848   Liu Y (2007). Preliminary Linkage Analysis of Keloid Susceptibility Loci and Polymorphisms of Correlation Genes in Chinese Han Population. Master's thesis, China Medical University, Shenyang.   Liu YB (2008). The Study of Impaired Apoptosis Function of Fas and P53 Protein in the Fibroblasts Derived from Keloid. PhD thesis, Southern Medical University, Guangzhou.   Liu YB, Gao JH, Duan HJ and Liu XJ (2003). Investigation of p53 gene mutations in keloids using PCR-SSCP. Zhonghua Zheng Xing Wai Ke Za Zhi 19: 258-260. PMid:14628411   Liu W, Jiang YH, Li YL, Lin ZH, et al. (2004). Experimental study on p53 gene mutation in keloid fibroblasts. Zhonghua Shao Shang Za Zhi 20: 85-87. PMid:15312469   Marneros AG and Krieg T (2004). Keloids-clinical diagnosis, pathogenesis, and treatment options. J. Dtsch. Dermatol. Ges. 2: 905-913. http://dx.doi.org/10.1046/j.1439-0353.2004.04077.x PMid:16281608   Matlashewski GJ, Tuck S, Pim D, Lamb P, et al. (1987). Primary structure polymorphism at amino acid residue 72 of human p53. Mol. Cell Biol. 7: 961-963. PMid:3547088 PMCid:365159   McGregor JM, Harwood CA, Brooks L, Fisher SA, et al. (2002). Relationship between p53 codon 72 polymorphism and susceptibility to sunburn and skin cancer. J. Invest. Dermatol. 119: 84-90. http://dx.doi.org/10.1046/j.1523-1747.2002.01655.x PMid:12164929   Menezes HL, Juca MJ, Gomes EG, Nunes BL, et al. (2010). Analysis of the immunohistochemical expressions of p53, bcl-2 and Ki-67 in colorectal adenocarcinoma and their correlations with the prognostic factors. Arq. Gastroenterol. 47: 141-147. http://dx.doi.org/10.1590/S0004-28032010000200005 PMid:20721457   Peters JL, Sutton AJ, Jones DR, Abrams KR, et al. (2006). Comparison of two methods to detect publication bias in meta-analysis. JAMA 295: 676-680. http://dx.doi.org/10.1001/jama.295.6.676 PMid:16467236   Pezeshki A, Sari-Aslani F, Ghaderi A and Doroudchi M (2006). p53 codon 72 polymorphism in basal cell carcinoma of the skin. Pathol. Oncol. Res. 12: 29-33. http://dx.doi.org/10.1007/BF02893428 PMid:16554913   Saed GM, Ladin D, Olson J, Han X, et al. (1998). Analysis of p53 gene mutations in keloids using polymerase chain reaction-based single-strand conformational polymorphism and DNA sequencing. Arch. Dermatol. 134: 963-967. http://dx.doi.org/10.1001/archderm.134.8.963 PMid:9722726   Sakamuro D, Sabbatini P, White E and Prendergast GC (1997). The polyproline region of p53 is required to activate apoptosis but not growth arrest. Oncogene 15: 887-898. http://dx.doi.org/10.1038/sj.onc.1201263 PMid:9285684   Sayah DN, Soo C, Shaw WW, Watson J, et al. (1999). Downregulation of apoptosis-related genes in keloid tissues. J. Surg. Res. 87: 209-216. http://dx.doi.org/10.1006/jsre.1999.5761 PMid:10600351   Sjalander A, Birgander R, Kivela A and Beckman G (1995). p53 polymorphisms and haplotypes in different ethnic groups. Hum. Hered. 45: 144-149. http://dx.doi.org/10.1159/000154275 PMid:7615299   Tanaka A, Hatoko M, Tada H, Iioka H, et al. (2004). Expression of p53 family in scars. J. Dermatol. Sci. 34: 17-24. http://dx.doi.org/10.1016/j.jdermsci.2003.09.005 PMid:14757278   Teofoli P, Barduagni S, Ribuffo M, Campanella A, et al. (1999). Expression of Bcl-2, p53, c-jun and c-fos protooncogenes in keloids and hypertrophic scars. J. Dermatol. Sci. 22: 31-37. http://dx.doi.org/10.1016/S0923-1811(99)00040-7   Thomas M, Kalita A, Labrecque S, Pim D, et al. (1999). Two polymorphic variants of wild-type p53 differ biochemically and biologically. Mol. Cell Biol. 19: 1092-1100. PMid:9891044 PMCid:116039   Vandenbroucke JP, von Elm E, Altman DG, Gotzsche PC, et al. (2007). Strengthening the Reporting of Observational Studies in Epidemiology (STROBE): explanation and elaboration. Epidemiology 18: 805-835. http://dx.doi.org/10.1097/EDE.0b013e3181577511 PMid:18049195   Walker KK and Levine AJ (1996). Identification of a novel p53 functional domain that is necessary for efficient growth suppression. Proc. Natl. Acad. Sci. U. S. A. 93: 15335-15340. http://dx.doi.org/10.1073/pnas.93.26.15335 PMid:8986812 PMCid:26405   Wang CM, Hiko H and Nakazawa N (2005). Investigation of p53 polymorphism for genetic predisposition of keloid and hypertrophic scar. Zhonghua Zheng Xing Wai Ke Za Zhi 21: 32-35. PMid:15844595   Yan L, Lu XY, Wang CM, Cao R, et al. (2007). Association between p53 gene codon 72 polymorphism and keloid in Chinese population. Zhonghua Zheng Xing Wai Ke Za Zhi 23: 428-430. PMid:18161363   Zhuo Y, Gao JH, Luo SQ, Zeng WS, et al. (2005). p53 gene codon 72 polymorphism and susceptibility to keloid. Zhonghua Zheng Xing Wai Ke Za Zhi 21: 201-203. PMid:16128105   Zhuo Y, Gao JH and Zeng XY (2008). The application of P53 gene detection kit for susceptibility of keloid. Zhongguo Mei Rong Yi Xue 5: 694-696.   Zintzaras E and Ioannidis JP (2005). Heterogeneity testing in meta-analysis of genome searches. Genet. Epidemiol. 28: 123-137. http://dx.doi.org/10.1002/gepi.20048 PMid:15593093
Y. J. Luo, Gao, W. X., Li, S. Z., Huang, X. W., Chen, Y., Liu, F. Y., Huang, Q. Y., and Gao, Y. Q., Mitochondrial haplogroup D4 confers resistance and haplogroup B is a genetic risk factor for high-altitude pulmonary edema among Han Chinese, vol. 11, pp. 3658-3667, 2012.
Ahsan A, Mohd G, Norboo T, Baig MA, et al. (2006). Heterozygotes of NOS3 polymorphisms contribute to reduced nitrogen oxides in high-altitude pulmonary edema. Chest 130: 1511-1519. http://dx.doi.org/10.1378/chest.130.5.1511 PMid:17099031   Alexe G, Fuku N, Bilal E, Ueno H, et al. (2007). Enrichment of longevity phenotype in mtDNA haplogroups D4b2b, D4a, and D5 in the Japanese population. Hum. Genet. 121: 347-356. http://dx.doi.org/10.1007/s00439-007-0330-6 PMid:17308896   Andrews RM, Kubacka I, Chinnery PF, Lightowlers RN, et al. (1999). Reanalysis and revision of the Cambridge reference sequence for human mitochondrial DNA. Nat. Genet. 23: 147. http://dx.doi.org/10.1038/13779 PMid:10508508   Barany F (1991). Genetic disease detection and DNA amplification using cloned thermostable ligase. Proc. Natl. Acad. Sci. U. S. A. 88: 189-193. http://dx.doi.org/10.1073/pnas.88.1.189 PMid:1986365 PMCid:50775   Bartsch P, Mairbaurl H, Maggiorini M and Swenson ER (2005). Physiological aspects of high-altitude pulmonary edema. J. Appl. Physiol. 98: 1101-1110. http://dx.doi.org/10.1152/japplphysiol.01167.2004 PMid:15703168   Basnyat B and Murdoch DR (2003). High-altitude illness. Lancet 361: 1967-1974. http://dx.doi.org/10.1016/S0140-6736(03)13591-X   Bilal E, Rabadan R, Alexe G, Fuku N, et al. (2008). Mitochondrial DNA haplogroup D4a is a marker for extreme longevity in Japan. PLoS One 3: e2421. http://dx.doi.org/10.1371/journal.pone.0002421 PMid:18545700 PMCid:2408726   Brandon MC, Ruiz-Pesini E, Mishmar D, Procaccio V, et al. (2009). MITOMASTER: a bioinformatics tool for the analysis of mitochondrial DNA sequences. Hum. Mutat. 30: 1-6. http://dx.doi.org/10.1002/humu.20801 PMid:18566966   Burk A, Douzery EJP and Springer MS (2002). The secondary structure of mammalian mitochondrial 16S rRNA molecules: refinements based on a comparative phylogenetic approach. J. Mamm. Evol. 9: 225-252. http://dx.doi.org/10.1023/A:1022649516930   Castello PR, David PS, McClure T, Crook Z, et al. (2006). Mitochondrial cytochrome oxidase produces nitric oxide under hypoxic conditions: implications for oxygen sensing and hypoxic signaling in eukaryotes. Cell Metab. 3: 277-287. http://dx.doi.org/10.1016/j.cmet.2006.02.011 PMid:16581005   Chandel NS, Maltepe E, Goldwasser E, Mathieu CE, et al. (1998). Mitochondrial reactive oxygen species trigger hypoxia-induced transcription. Proc. Natl. Acad. Sci. U. S. A. 95: 11715-11720. http://dx.doi.org/10.1073/pnas.95.20.11715 PMid:9751731 PMCid:21706   Dada LA, Chandel NS, Ridge KM, Pedemonte C, et al. (2003). Hypoxia-induced endocytosis of Na,K-ATPase in alveolar epithelial cells is mediated by mitochondrial reactive oxygen species and PKC-zeta. J. Clin. Invest. 111: 1057-1064. PMid:12671055 PMCid:152585   Dehnert C, Berger MM, Mairbaurl H and Bartsch P (2007). High altitude pulmonary edema: a pressure-induced leak. Respir. Physiol. Neurobiol. 158: 266-273. http://dx.doi.org/10.1016/j.resp.2007.05.002 PMid:17602898   Droma Y, Hanaoka M, Ota M, Katsuyama Y, et al. (2002). Positive association of the endothelial nitric oxide synthase gene polymorphisms with high-altitude pulmonary edema. Circulation 106: 826-830. http://dx.doi.org/10.1161/01.CIR.0000024409.30143.70 PMid:12176955   Guo LJ, Oshida Y, Fuku N, Takeyasu T, et al. (2005). Mitochondrial genome polymorphisms associated with type-2 diabetes or obesity. Mitochondrion 5: 15-33. http://dx.doi.org/10.1016/j.mito.2004.09.001 PMid:16060290   Hanaoka M, Droma Y, Hotta J, Matsuzawa Y, et al. (2003). Polymorphisms of the tyrosine hydroxylase gene in subjects susceptible to high-altitude pulmonary edema. Chest 123: 54-58. http://dx.doi.org/10.1378/chest.123.1.54 PMid:12527603   Hanaoka M, Droma Y, Ota M, Ito M, et al. (2009). Polymorphisms of human vascular endothelial growth factor gene in high-altitude pulmonary oedema susceptible subjects. Respirology 14: 46-52. http://dx.doi.org/10.1111/j.1440-1843.2008.01420.x PMid:19144048   Hochachka PW (1986). Defense strategies against hypoxia and hypothermia. Science 231: 234-241. http://dx.doi.org/10.1126/science.2417316 PMid:2417316   Hotta J, Hanaoka M, Droma Y, Katsuyama Y, et al. (2004). Polymorphisms of renin-angiotensin system genes with high-altitude pulmonary edema in Japanese subjects. Chest 126: 825-830. http://dx.doi.org/10.1378/chest.126.3.825 PMid:15364762   Hultgren HN and Marticorena EA (1978). High altitude pulmonary edema. Epidemiologic observations in Peru. Chest 74: 372-376. http://dx.doi.org/10.1378/chest.74.4.372 PMid:699645   Kivisild T, Tolk HV, Parik J, Wang Y, et al. (2002). The emerging limbs and twigs of the East Asian mtDNA tree. Mol. Biol. Evol. 19: 1737-1751. http://dx.doi.org/10.1093/oxfordjournals.molbev.a003996 PMid:12270900   Kong QP, Bandelt HJ, Sun C, Yao YG, et al. (2006). Updating the East Asian mtDNA phylogeny: a prerequisite for the identification of pathogenic mutations. Hum. Mol. Genet. 15: 2076-2086. http://dx.doi.org/10.1093/hmg/ddl130 PMid:16714301   Kwast KE, Burke PV, Staahl BT and Poyton RO (1999). Oxygen sensing in yeast: evidence for the involvement of the respiratory chain in regulating the transcription of a subset of hypoxic genes. Proc. Natl. Acad. Sci. U. S. A. 96: 5446-5451. http://dx.doi.org/10.1073/pnas.96.10.5446 PMid:10318903 PMCid:21879   Luo Y, Tang S, Gao W, Chen L, et al. (2010). Genotyping mitochondrial DNA single nucleotide polymorphisms by PCR ligase detection reactions. Clin. Chem. Lab. Med. 48: 475-483. http://dx.doi.org/10.1515/cclm.2010.097 PMid:20128730   Mortimer H, Patel S and Peacock AJ (2004). The genetic basis of high-altitude pulmonary oedema. Pharmacol. Ther. 101: 183-192. http://dx.doi.org/10.1016/j.pharmthera.2003.11.003 PMid:14761704   Nigou M, Parfait B, Clauser E and Olivier JL (1998). Detection and quantification of the A3243G mutation of mitochondrial DNA by ligation detection reaction. Mol. Cell Probes 12: 273-282. http://dx.doi.org/10.1006/mcpr.1998.0191 PMid:9778452   Nishigaki Y, Yamada Y, Fuku N, Matsuo H, et al. (2007). Mitochondrial haplogroup N9b is protective against myocardial infarction in Japanese males. Hum. Genet. 120: 827-836. http://dx.doi.org/10.1007/s00439-006-0269-z PMid:17033820   Peacock AJ (1995). High altitude pulmonary oedema: who gets it and why? Eur. Respir. J. 8: 1819-1821. http://dx.doi.org/10.1183/09031936.95.08111819 PMid:8620944   Qi Y, Niu W, Zhu T, Zhou W, et al. (2008). Synergistic effect of the genetic polymorphisms of the renin-angiotensin-aldosterone system on high-altitude pulmonary edema: a study from Qinghai-Tibet altitude. Eur. J. Epidemiol. 23: 143-152. http://dx.doi.org/10.1007/s10654-007-9208-0 PMid:17987391   Qi Y, Niu WQ, Zhu TC, Liu JL, et al. (2009). Genetic interaction of Hsp70 family genes polymorphisms with high-altitude pulmonary edema among Chinese railway constructors at altitudes exceeding 4000 meters. Clin. Chim. Acta 405: 17-22. http://dx.doi.org/10.1016/j.cca.2009.03.056 PMid:19351530   Rosa A, Fonseca BV, Krug T, Manso H, et al. (2008). Mitochondrial haplogroup H1 is protective for ischemic stroke in Portuguese patients. BMC Med. Genet. 9: 57. http://dx.doi.org/10.1186/1471-2350-9-57 PMid:18593462 PMCid:2492853   Sarada S, Himadri P, Mishra C, Geetali P, et al. (2008). Role of oxidative stress and NFkB in hypoxia-induced pulmonary edema. Exp. Biol. Med. 233: 1088-1098. http://dx.doi.org/10.3181/0712-RM-337 PMid:18641050   Sartori C, Allemann Y and Scherrer U (2007). Pathogenesis of pulmonary edema: learning from high-altitude pulmonary edema. Respir. Physiol. Neurobiol. 159: 338-349. http://dx.doi.org/10.1016/j.resp.2007.04.006 PMid:17532272   Saxena S, Kumar R, Madan T, Gupta V, et al. (2005). Association of polymorphisms in pulmonary surfactant protein A1 and A2 genes with high-altitude pulmonary edema. Chest 128: 1611-1619. http://dx.doi.org/10.1378/chest.128.3.1611 PMid:16162765   Schurr TG and Wallace DC (2002). Mitochondrial DNA diversity in Southeast Asian populations. Hum. Biol. 74: 431-452. http://dx.doi.org/10.1353/hub.2002.0034 PMid:12180765   Wallace DC (2005). A mitochondrial paradigm of metabolic and degenerative diseases, aging, and cancer: a dawn for evolutionary medicine. Annu. Rev. Genet. 39: 359-407. http://dx.doi.org/10.1146/annurev.genet.39.110304.095751 PMid:16285865 PMCid:2821041   Wen B, Li H, Lu D, Song X, et al. (2004). Genetic evidence supports demic diffusion of Han culture. Nature 431: 302-305. http://dx.doi.org/10.1038/nature02878 PMid:15372031   Yao YG, Kong QP, Bandelt HJ, Kivisild T, et al. (2002). Phylogeographic differentiation of mitochondrial DNA in Han Chinese. Am. J. Hum. Genet. 70: 635-651. http://dx.doi.org/10.1086/338999 PMid:11836649 PMCid:384943   Yoshida T and Tuder RM (2007). Pathobiology of cigarette smoke-induced chronic obstructive pulmonary disease. Physiol. Rev. 87: 1047-1082. http://dx.doi.org/10.1152/physrev.00048.2006 PMid:17615396