Publications
Found 2 results
Filters: Author is P.C. Venere [Clear All Filters]
,
“Lack of association of CYP1A1-MspI SNP and GSTM1 null genotypes with cancer in a Brazilian family with unusually high cancer incidence”, vol. 11, pp. 1610-1617, 2012.
,
Arruda VR, Grignolli CE, Goncalves MS, Soares MC, et al. (1998). Prevalence of homozygosity for the deleted alleles of glutathione S-transferase mu (GSTM1) and theta (GSTT1) among distinct ethnic groups from Brazil: relevance to environmental carcinogenesis? Clin. Genet. 54: 210-214.
PMid:9788723
Arvanitis DA, Koumantakis GE, Goumenou AG, Matalliotakis IM, et al. (2003). CYP1A1, CYP19, and GSTM1 polymorphisms increase the risk of endometriosis. Fertil. Steril. 79 (Suppl 1): 702-709.
http://dx.doi.org/10.1016/S0015-0282(02)04817-3
Autrup H (2000). Genetic polymorphisms in human xenobiotica metabolizing enzymes as susceptibility factors in toxic response. Mutat. Res. 464: 65-76.
http://dx.doi.org/10.1016/S1383-5718(99)00167-9
Cha IH, Park JY, Chung WY, Choi MA, et al. (2007). Polymorphisms of CYP1A1 and GSTM1 genes and susceptibility to oral cancer. Yonsei Med. J. 48: 233-239.
http://dx.doi.org/10.3349/ymj.2007.48.2.233
PMid:17461521 PMCid:2628133
Deakin M, Elder J, Hendrickse C, Peckham D, et al. (1996). Glutathione S-transferase GSTT1 genotypes and susceptibility to cancer: studies of interactions with GSTM1 in lung, oral, gastric and colorectal cancers. Carcinogenesis 17: 881-884.
http://dx.doi.org/10.1093/carcin/17.4.881
PMid:8625505
Duarte RLM and Paschoal MEM (2005). Molecular markers in lung cancer: prognostic role and relationship to smoking. J. Bras. Pneumol. 32: 56-65.
http://dx.doi.org/10.1590/S1806-37132006000100012
Eaton DL and Bammler TK (1999). Concise review of the glutathione S-transferases and their significance to toxicology. Toxicol. Sci. 49: 156-164.
http://dx.doi.org/10.1093/toxsci/49.2.156
PMid:10416260
Gaspar P, Moreira J, Kvitko K, Torres M, et al. (2004). CYP1A1, CYP2E1, GSTM1, GSTT1, GSTP1, and TP53 polymorphisms: do they indicate susceptibility to chronic obstructive pulmonary disease and non-small-cell lung cancer? Genet. Mol. Biol. 27: 133-138.
http://dx.doi.org/10.1590/S1415-47572004000200001
González A, Ramírez V, Cuenca P and Sierra R (2004). Polimorfismos en los genes de desintoxicación CYP1A1, CYP2E1, GSTT1 y GSTM1 en la susceptibilidad al cáncer gástrico. Rev. Biol. Trop. 52: 591-600.
PMid:17361553
Hatagima A (2002). Genetic polymorphisms and metabolism of endocrine disruptors in cancer susceptibility. Cad. Saúde Pública 18: 357-377.
http://dx.doi.org/10.1590/S0102-311X2002000200002
PMid:11923879
Hatagima A, Klautau-Guimarães MN, Silva FP and Cabello PH (2000). Glutathione S-transferase M1 (GSTM1) polymorphism in two Brazilian populations. Genet. Mol. Biol. 23: 709-713.
http://dx.doi.org/10.1590/S1415-47572000000400003
Hildebrand CE, Gonzalez FJ, Mcbride OW and Nebert DW (1985). Assignment of the human 2,3,7,8-tetrachlorodibenzo-p-dioxin-inducible cytochrome P1-450 gene to chromosome 15. Nucleic Acids Res. 13: 2009-2016.
http://dx.doi.org/10.1093/nar/13.6.2009
PMid:4000952 PMCid:341131
Hirvonen A (1995). Genetic factors in individual responses to environmental exposures. J. Occup. Environ. Med. 37: 37-43.
http://dx.doi.org/10.1097/00043764-199501000-00006
PMid:7620941
Hirvonen A, Husgafvel-Pursiainen K, Anttila S and Vainio H (1993). The GSTM1 null genotype as a potential risk modifier for squamous cell carcinoma of the lung. Carcinogenesis 14: 1479-1481.
http://dx.doi.org/10.1093/carcin/14.7.1479
PMid:8330369
Kawajiri K, Watanabe J, Gotoh O, Tagashira Y, et al. (1986). Structure and drug inducibility of the human cytochrome P-450c gene. Eur. J. Biochem. 159: 219-225.
http://dx.doi.org/10.1111/j.1432-1033.1986.tb09857.x
PMid:3019683
Lahiri DK and Nurnberger JI Jr (1991). A rapid non-enzymatic method for the preparation of HMW DNA from blood for RFLP studies. Nucleic Acids Res. 19: 5444.
http://dx.doi.org/10.1093/nar/19.19.5444
PMid:1681511 PMCid:328920
Landi MT, Bertazzi PA, Shields PG, Clark G, et al. (1994). Association between CYP1A1 genotype, mRNA expression and enzymatic activity in humans. Pharmacogenetics 4: 242-246.
http://dx.doi.org/10.1097/00008571-199410000-00002
PMid:7894496
Lin HJ, Han CY, Bernstein DA, Hsiao W, et al. (1994). Ethnic distribution of the glutathione transferase Mu 1-1 (GSTM1) null genotype in 1473 individuals and application to bladder cancer susceptibility. Carcinogenesis 15: 1077-1081.
http://dx.doi.org/10.1093/carcin/15.5.1077
PMid:8200072
Maciel ME, Oliveira FK, Propst GB, da Graca BM, et al. (2009). Population analysis of xenobiotic metabolizing genes in South Brazilian Euro and Afro-descendants. Genet. Mol. Biol. 32: 723-728.
http://dx.doi.org/10.1590/S1415-47572009005000087
PMid:21637445 PMCid:3036891
Miller MS, McCarver DG, Bell DA, Eaton DL, et al. (1997). Genetic polymorphisms in human drug metabolic enzymes. Fund. Appl. Toxicol. 40: 1-14.
http://dx.doi.org/10.1006/faat.1997.2382
PMid:9398483
Nakata LC, Goloni-Bertollo EM, Santos I and Oliani AH (2004). Biomarcadores de suscetibilidade à endometriose. Rev. Bras. Ginecol. Obstet. 26: 299-304.
http://dx.doi.org/10.1590/S0100-72032004000400006
Oga S, Camargo MMA and Batistuzzo JAO (2003). Fundamentos de Toxicologia. 2ª ed. Atheneu, São Paulo.
Rebbeck TR (1997). Molecular epidemiology of the human glutathione S-transferase genotypes GSTM1 and GSTT1 in cancer susceptibility. Cancer Epidemiol. Biomarkers Prev. 6: 733-743.
PMid:9298582
Rossini A, Rapozo DC, Amorim LM, Macedo JM, et al. (2002). Frequencies of GSTM1, GSTT1, and GSTP1 polymorphisms in a Brazilian population. Genet. Mol. Res. 1: 233-240.
PMid:14963830
Santella RM, Perera FP, Young TL, Zhang YJ, et al. (1995). Polycyclic aromatic hydrocarbon-DNA and protein adducts in coal tar treated patients and controls and their relationship to glutathione S-transferase genotype. Mutat. Res. 334: 117-124.
http://dx.doi.org/10.1016/0165-1161(95)90001-2
Sarmanová J, Benesová K, Gut I, Nedelcheva-Kristensen V, et al. (2001). Genetic polymorphisms of biotransformation enzymes in patients with Hodgkin's and non-Hodgkin's lymphomas. Hum. Mol. Genet. 10: 1265-1273.
http://dx.doi.org/10.1093/hmg/10.12.1265
PMid:11406608
Sato M, Sato T, Izumo T and Amagasa T (1999). Genetic polymorphism of drug metabolizing enzymes and susceptibility to oral cancer. Carcinogenesis 20: 1927-1931.
http://dx.doi.org/10.1093/carcin/20.10.1927
PMid:10506106
Schnakenberg E, Fabig KR, Stanulla M, Strobl N, et al. (2007). A cross-sectional study of self-reported chemical-related sensitivity is associated with gene variants of drug-metabolizing enzymes. Environ. Health 6: 6.
http://dx.doi.org/10.1186/1476-069X-6-6
PMid:17291352 PMCid:1802749
Sugimura T (2000). Nutrition and dietary carcinogens. Carcinogenesis 21: 387-395.
http://dx.doi.org/10.1093/carcin/21.3.387
PMid:10688859
Vineis P and Perera F (2007). Molecular epidemiology and biomarkers in etiologic cancer research: the new in light of the old. Cancer Epidemiol. Biomarkers Prev. 16: 1954-1965.
http://dx.doi.org/10.1158/1055-9965.EPI-07-0457
PMid:17932342
Wang Z, Wilson GF and Griffith LC (2002). Calcium/calmodulin-dependent protein kinase II phosphorylates and regulates the Drosophila eag potassium channel. J. Biol. Chem. 277: 24022-24029.
http://dx.doi.org/10.1074/jbc.M201949200
PMid:11980904
Zhong S, Wyllie AH, Barnes D, Wolf CR, et al. (1993). Relationship between the GSTM1 genetic polymorphism and susceptibility to bladder, breast and colon cancer. Carcinogenesis 14: 1821-1824.
http://dx.doi.org/10.1093/carcin/14.9.1821
PMid:8403204