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2012
Y. Wang, Zhou, X. O., Zhang, Y., Gao, P. J., and Zhu, D. L., Association of the CD36 gene with impaired glucose tolerance, impaired fasting glucose, type-2 diabetes, and lipid metabolism in essential hypertensive patients, vol. 11, pp. 2163-2170, 2012.
Aitman TJ, Glazier AM, Wallace CA, Cooper LD, et al. (1999). Identification of Cd36 (Fat) as an insulin-resistance gene causing defective fatty acid and glucose metabolism in hypertensive rats. Nat. Genet. 21: 76-83. http://dx.doi.org/10.1038/5013 PMid:9916795   Almgren T, Wilhelmsen L, Samuelsson O, Himmelmann A, et al. (2007). Diabetes in treated hypertension is common and carries a high cardiovascular risk: results from a 28-year follow-up. J. Hypertens. 25: 1311-1317. http://dx.doi.org/10.1097/HJH.0b013e328122dd58 PMid:17563546   Bokor S, Legry V, Meirhaeghe A, Ruiz JR, et al. (2010). Single-nucleotide polymorphism of CD36 locus and obesity in European adolescents. Obesity 18: 1398-1403. http://dx.doi.org/10.1038/oby.2009.412 PMid:19893500   Coburn CT, Knapp FF Jr, Febbraio M, Beets AL, et al. (2000). Defective uptake and utilization of long chain fatty acids in muscle and adipose tissues of CD36 knockout mice. J. Biol. Chem. 275: 32523-32529. http://dx.doi.org/10.1074/jbc.M003826200 PMid:10913136   Gurnell M, Savage DB, Chatterjee VK and O'Rahilly S (2003). The metabolic syndrome: peroxisome proliferator-activated receptor gamma and its therapeutic modulation. J. Clin. Endocrinol. Metab. 88: 2412-2421. http://dx.doi.org/10.1210/jc.2003-030435 PMid:12788836   Hajri T and Abumrad NA (2002). Fatty acid transport across membranes: relevance to nutrition and metabolic pathology. Annu. Rev. Nutr. 22: 383-415. http://dx.doi.org/10.1146/annurev.nutr.22.020402.130846 PMid:12055351   Han XX, Chabowski A, Tandon NN, Calles-Escandon J, et al. (2007). Metabolic challenges reveal impaired fatty acid metabolism and translocation of FAT/CD36 but not FABPpm in obese Zucker rat muscle. Am. J. Physiol. Endocrinol. Metab. 293: E566-E575. http://dx.doi.org/10.1152/ajpendo.00106.2007 PMid:17519284   Harasim E, Kalinowska A, Chabowski A and Stepek T (2008). The role of fatty-acid transport proteins (FAT/CD36, FABPpm, FATP) in lipid metabolism in skeletal muscles. Postepy Hig. Med. Dosw. 62: 433-441.   Lepretre F, Vasseur F, Vaxillaire M, Scherer PE, et al. (2004). A CD36 nonsense mutation associated with insulin resistance and familial type 2 diabetes. Hum. Mutat. 24: 104. http://dx.doi.org/10.1002/humu.9256 PMid:15221799   Love-Gregory L, Sherva R, Sun L, Wasson J, et al. (2008). Variants in the CD36 gene associate with the metabolic syndrome and high-density lipoprotein cholesterol. Hum. Mol. Genet. 17: 1695-1704. http://dx.doi.org/10.1093/hmg/ddn060 PMid:18305138 PMCid:2655228   Ma X, Bacci S, Mlynarski W, Gottardo L, et al. (2004). A common haplotype at the CD36 locus is associated with high free fatty acid levels and increased cardiovascular risk in Caucasians. Hum. Mol. Genet. 13: 2197-2205. http://dx.doi.org/10.1093/hmg/ddh233 PMid:15282206   Noel SE, Lai CQ, Mattei J, Parnell LD, et al. (2010). Variants of the CD36 gene and metabolic syndrome in Boston Puerto Rican adults. Atherosclerosis 211: 210-215. http://dx.doi.org/10.1016/j.atherosclerosis.2010.02.009 PMid:20223461 PMCid:2923842   Osei K, Rhinesmith S, Gaillard T and Schuster D (2004). Impaired insulin sensitivity, insulin secretion, and glucose effectiveness predict future development of impaired glucose tolerance and type 2 diabetes in pre-diabetic African Americans: implications for primary diabetes prevention. Diabetes Care 27: 1439-1446. http://dx.doi.org/10.2337/diacare.27.6.1439 PMid:15161801   Pontiroli AE, Pizzocri P, Caumo A, Perseghin G, et al. (2004). Evaluation of insulin release and insulin sensitivity through oral glucose tolerance test: differences between NGT, IFG, IGT, and type 2 diabetes mellitus. A cross-sectional and follow-up study. Acta Diabetol. 41: 70-76. http://dx.doi.org/10.1007/s00592-004-0147-x PMid:15224208   Pravenec M and Kurtz TW (2002). Genetics of Cd36 and the hypertension metabolic syndrome. Semin. Nephrol. 22: 148-153. http://dx.doi.org/10.1053/snep.2002.2002.30218 PMid:11891508   Susztak K, Ciccone E, McCue P, Sharma K, et al. (2005). Multiple metabolic hits converge on CD36 as novel mediator of tubular epithelial apoptosis in diabetic nephropathy. PLoS Med. 2: e45. http://dx.doi.org/10.1371/journal.pmed.0020045 PMid:15737001 PMCid:549593   Wang X and Snieder H (2010). Genome-wide association studies and beyond: what's next in blood pressure genetics? Hypertension 56: 1035-1037. http://dx.doi.org/10.1161/HYPERTENSIONAHA.110.157214 PMid:21060002   Yamauchi T, Hara K, Maeda S, Yasuda K, et al. (2010). A genome-wide association study in the Japanese population identifies susceptibility loci for type 2 diabetes at UBE2E2 and C2CD4A-C2CD4B. Nat. Genet. 42: 864-868. http://dx.doi.org/10.1038/ng.660 PMid:20818381   Zhou X, Wang Y, Zhang Y, Gao P, et al. (2010). Association of CAPN10 gene with insulin sensitivity, glucose tolerance and renal function in essential hypertensive patients. Clin. Chim. Acta 411: 1126-1131. http://dx.doi.org/10.1016/j.cca.2010.04.012 PMid:20406624
2011
Y. Wang, Zhou, X. O., Zhang, Y., Gao, P. J., and Zhu, D. L., Association of KCNJ11 with impaired glucose regulation in essential hypertension, vol. 10, pp. 1111-1119, 2011.
Cederholm J and Wibell L (1990). Insulin release and peripheral sensitivity at the oral glucose tolerance test. Diabetes Res. Clin. Pract. 10: 167-175. doi:10.1016/0168-8227(90)90040-Z De Marco M, de Simone G, Roman MJ, Chinali M, et al. (2009). Cardiovascular and metabolic predictors of progression of prehypertension into hypertension: the strong heart study. Hypertension 54: 974-980. doi:10.1161/HYPERTENSIONAHA.109.129031 PMid:19720957    PMCid:2776057 Dudbridge F (2003). Pedigree disequilibrium tests for multilocus haplotypes. Genet. Epidemiol. 25: 115-121. doi:10.1002/gepi.10252 PMid:12916020 Florez JC, Jablonski KA, Kahn SE, Franks PW, et al. (2007). Type 2 diabetes-associated missense polymorphisms KCNJ11 E23K and ABCC8 A1369S influence progression to diabetes and response to interventions in the Diabetes Prevention Program. Diabetes 56: 531-536. doi:10.2337/db06-0966 PMid:17259403    PMCid:2267937 Gloyn AL, Pearson ER, Antcliff JF, Proks P, et al. (2004). Activating mutations in the gene encoding the ATP-sensitive potassium-channel subunit Kir6.2 and permanent neonatal diabetes. N. Engl. J. Med. 350: 1838-1849. doi:10.1056/NEJMoa032922 PMid:15115830 Hackam DG, Khan NA, Hemmelgarn BR, Rabkin SW, et al. (2010). The 2010 Canadian Hypertension Education Program recommendations for the management of hypertension: part 2 - therapy. Can. J. Cardiol. 26: 249-258. doi:10.1016/S0828-282X(10)70379-2 Lin YW, Bushman JD, Yan FF, Haidar S, et al. (2008). Destabilization of ATP-sensitive potassium channel activity by novel KCNJ11 mutations identified in congenital hyperinsulinism. J. Biol. Chem. 283: 9146-9156. doi:10.1074/jbc.M708798200 PMid:18250167    PMCid:2431039 Matthews DR, Hosker JP, Rudenski AS, Naylor BA, et al. (1985). Homeostasis model assessment: insulin resistance and beta-cell function from fasting plasma glucose and insulin concentrations in man. Diabetologia 28: 412-419. doi:10.1007/BF00280883 PMid:3899825 Nielsen EM, Hansen L, Carstensen B, Echwald SM, et al. (2003). The E23K variant of Kir6.2 associates with impaired post-OGTT serum insulin response and increased risk of type 2 diabetes. Diabetes 52: 573-577. doi:10.2337/diabetes.52.2.573 Ryder E, Gomez ME, Fernandez V, Campos G, et al. (2003). Presence of impaired insulin secretion and insulin resistance in normoglycemic male subjects with family history of type 2 diabetes. Diabetes Res. Clin. Pract. 60: 95-103. doi:10.1016/S0168-8227(02)00282-6 Saxena R, Voight BF, Lyssenko V, Burtt NP, et al. (2007). Genome-wide association analysis identifies loci for type 2 diabetes and triglyceride levels. Science 316: 1331-1336. doi:10.1126/science.1142358 PMid:17463246 Schaid DJ, Rowland CM, Tines DE, Jacobson RM, et al. (2002). Score tests for association between traits and haplotypes when linkage phase is ambiguous. Am. J. Hum. Genet. 70: 425-434. doi:10.1086/338688 PMid:11791212 Scott LJ, Mohlke KL, Bonnycastle LL, Willer CJ, et al. (2007). A genome-wide association study of type 2 diabetes in Finns detects multiple susceptibility variants. Science 316: 1341-1345. doi:10.1126/science.1142382 PMid:17463248 Seino S, Iwanaga T, Nagashima K and Miki T (2000). Diverse roles of K(ATP) channels learned from Kir6.2 genetically engineered mice. Diabetes 49: 311-318. doi:10.2337/diabetes.49.3.311 PMid:10868950 Vaxillaire M, Veslot J, Dina C, Proenca C, et al. (2008). Impact of common type 2 diabetes risk polymorphisms in the DESIR prospective study. Diabetes 57: 244-254. doi:10.2337/db07-0615 PMid:17977958 Villareal DT, Koster JC, Robertson H, Akrouh A, et al. (2009). Kir6.2 variant E23K increases ATP-sensitive K+ channel activity and is associated with impaired insulin release and enhanced insulin sensitivity in adults with normal glucose tolerance. Diabetes 58: 1869-1878. doi:10.2337/db09-0025 PMid:19491206    PMCid:2712777 Vlasakova Z, Pelikanova T, Karasova L and Skibova J (2004). Insulin secretion, sensitivity, and metabolic profile of young healthy offspring of hypertensive parents. Metabolism 53: 469-475. doi:10.1016/j.metabol.2003.10.030 PMid:15045694 Yokoi N, Kanamori M, Horikawa Y, Takeda J, et al. (2006). Association studies of variants in the genes involved in pancreatic beta-cell function in type 2 diabetes in Japanese subjects. Diabetes 55: 2379-2386. doi:10.2337/db05-1203 PMid:16873704 Zeggini E, Weedon MN, Lindgren CM, Frayling TM, et al. (2007). Replication of genome-wide association signals in UK samples reveals risk loci for type 2 diabetes. Science 316: 1336-1341. doi:10.1126/science.1142364 PMid:17463249