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“Influence of glutathione S-transferase polymorphisms on type-2 diabetes mellitus risk”, vol. 10, pp. 3722-3730, 2011.
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Ada AO, Suzen SH and Iscan M (2004). Polymorphisms of cytochrome P450 1A1, glutathione S-transferases M1 and T1 in a Turkish population. Toxicol. Lett. 151: 311-315.
http://dx.doi.org/10.1016/j.toxlet.2003.12.075
PMid:15177667
American Diabetes Association (2007). Diagnosis and classification of diabetes mellitus. Diabetes Care 30 (Suppl 1): 47.
Baynes JW (1991). Role of oxidative stress in development of complications in diabetes. Diabetes 40: 405-412.
http://dx.doi.org/10.2337/diabetes.40.4.405
PMid:2010041
Bessa SS, Ali EM and Hamdy SM (2009). The role of glutathione S- transferase M1 and T1 gene polymorphisms and oxidative stress-related parameters in Egyptian patients with essential hypertension. Eur. J. Intern. Med. 20: 625-630.
http://dx.doi.org/10.1016/j.ejim.2009.06.003
PMid:19782926
Bid HK, Konwar R, Saxena M, Chaudhari P, et al. (2010). Association of glutathione S-transferase (GSTM1, T1 and P1) gene polymorphisms with type 2 diabetes mellitus in north Indian population. J. Postgrad. Med. 56: 176-181.
http://dx.doi.org/10.4103/0022-3859.68633
PMid:20739761
D'Alo F, Voso MT, Guidi F, Massini G, et al. (2004). Polymorphisms of CYP1A1 and glutathione S-transferase and susceptibility to adult acute myeloid leukemia. Haematologica 89: 664-670.
PMid:15194533
Delles C, Padmanabhan S, Lee WK, Miller WH, et al. (2008). Glutathione S-transferase variants and hypertension. J. Hypertens. 26: 1343-1352.
http://dx.doi.org/10.1097/HJH.0b013e3282fe1d67
PMid:18551009
Doney AS, Lee S, Leese GP, Morris AD, et al. (2005). Increased cardiovascular morbidity and mortality in type 2 diabetes is associated with the glutathione S transferase theta-null genotype: a Go-DARTS study. Circulation 111: 2927-2934.
http://dx.doi.org/10.1161/CIRCULATIONAHA.104.509224
PMid:15927971
Douglas KT (1987). Mechanism of action of glutathione-dependent enzymes. Adv. Enzymol. Relat. Areas Mol. Biol. 59: 103-167.
PMid:2880477
Garte S, Gaspari L, Alexandrie AK, Ambrosone C, et al. (2001). Metabolic gene polymorphism frequencies in control populations. Cancer Epidemiol. Biomarkers Prev. 10: 1239-1248.
PMid:11751440
Gorogawa S, Kajimoto Y, Umayahara Y, Kaneto H, et al. (2002). Probucol preserves pancreatic beta-cell function through reduction of oxidative stress in type 2 diabetes. Diabetes Res. Clin. Pract. 57: 1-10.
http://dx.doi.org/10.1016/S0168-8227(02)00005-0
Gsur A, Haidinger G, Hinteregger S, Bernhofer G, et al. (2001). Polymorphisms of glutathione-S-transferase genes (GSTP1, GSTM1 and GSTT1) and prostate-cancer risk. Int. J. Cancer 95: 152-155.
http://dx.doi.org/10.1002/1097-0215(20010520)95:3<152::AID-IJC1026>3.0.CO;2-S
Hayes JD and McLellan LI (1999). Glutathione and glutathione-dependent enzymes represent a co-ordinately regulated defence against oxidative stress. Free Radic. Res. 31: 273-300.
http://dx.doi.org/10.1080/10715769900300851
PMid:10517533
Hayes JD and Strange RC (2000). Glutathione S-transferase polymorphisms and their biological consequences. Pharmacology 61: 154-166.
http://dx.doi.org/10.1159/000028396
PMid:10971201
Hori M, Oniki K, Ueda K, Goto S, et al. (2007). Combined glutathione S-transferase T1 and M1 positive genotypes afford protection against type 2 diabetes in Japanese. Pharmacogenomics 8: 1307-1314.
http://dx.doi.org/10.2217/14622416.8.10.1307
PMid:17979505
London SJ, Yuan JM, Chung FL, Gao YT, et al. (2000). Isothiocyanates, glutathione S-transferase M1 and T1 polymorphisms, and lung-cancer risk: a prospective study of men in Shanghai, China. Lancet 356: 724-729.
http://dx.doi.org/10.1016/S0140-6736(00)02631-3
Maritim AC, Sanders RA and Watkins JB, III (2003). Diabetes, oxidative stress, and antioxidants: a review. J. Biochem. Mol. Toxicol. 17: 24-38.
http://dx.doi.org/10.1002/jbt.10058
PMid:12616644
Pemble S, Schroeder KR, Spencer SR, Meyer DJ, et al. (1994). Human glutathione S-transferase theta (GSTT1): cDNA cloning and the characterization of a genetic polymorphism. Biochem. J. 300: 271-276.
PMid:8198545 PMCid:1138152
Shaikh RS, Amir M, Masood AI, Sohail A, et al. (2010). Frequency distribution of GSTM1 and GSTT1 null allele in Pakistani population and risk of disease incidence. Environ. Toxicol. Pharmacol. 30: 76-79.
http://dx.doi.org/10.1016/j.etap.2010.04.002
PMid:21787632
Shaw JE, Sicree RA and Zimmet PZ (2010). Global estimates of the prevalence of diabetes for 2010 and 2030. Diabetes Res. Clin. Pract. 87: 4-14.
http://dx.doi.org/10.1016/j.diabres.2009.10.007
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Sheehan D, Meade G, Foley VM and Dowd CA (2001). Structure, function and evolution of glutathione transferases: implications for classification of non-mammalian members of an ancient enzyme superfamily. Biochem. J. 360: 1-16.
http://dx.doi.org/10.1042/0264-6021:3600001
PMid:11695986 PMCid:1222196
Stephens JW, Khanolkar MP and Bain SC (2009). The biological relevance and measurement of plasma markers of oxidative stress in diabetes and cardiovascular disease. Atherosclerosis 202: 321-329.
http://dx.doi.org/10.1016/j.atherosclerosis.2008.06.006
PMid:18640680
Wang G, Zhang L and Li Q (2006). Genetic polymorphisms of GSTT1, GSTM1, and NQO1 genes and diabetes mellitus risk in Chinese population. Biochem. Biophys. Res. Commun. 341: 310-313.
http://dx.doi.org/10.1016/j.bbrc.2005.12.195
PMid:16413497
Wild S, Roglic G, Green A, Sicree R, et al. (2004). Global prevalence of diabetes: estimates for the year 2000 and projections for 2030. Diabetes Care 27: 1047-1053.
http://dx.doi.org/10.2337/diacare.27.5.1047
PMid:15111519